英语翻译Ferric iron is reduced in a reaction with superoxide anion and subsequently ferrousiron is oxidized and thus iron can generate highly reactivehydroxyl radicals in Fenton reaction.The summary reaction,referred to as Haber-Weiss reaction,re

英语翻译Ferric iron is reduced in a reaction with superoxide anion and subsequently ferrousiron is oxidized and thus iron can generate highly reactivehydroxyl radicals in Fenton reaction.The summary reaction,referred to as Haber-Weiss reaction,re
英语翻译
Ferric iron is reduced in a reaction with superoxide anion and subsequently ferrous
iron is oxidized and thus iron can generate highly reactive
hydroxyl radicals in Fenton reaction.The summary reaction,
referred to as Haber-Weiss reaction,results in
hydroxyl radical production and decrease in superoxide
concentration [15].Low-molecular-mass iron complexes
are also able to produce ROS [16,17].
We have demonstrated previously that iron deprivation
stimulates iron uptake from ferric citrate by human erythroleukemia
K562 cells and that this stimulation depends on
protein synthesis.However,no involvement of any of known
proteins with a suggested role in non-transferrin iron transport
across plasma membrane was detected [18].The existence of
transport system for iron uptake from low-molecular-mass
complexes including ferric citrate was well documented [19–
23].However,specific molecules involved in the transport
system remain obscure in spite of the fact that the identification
of these molecules is highly challenging mainly due to
their potential involvement in some iron disorders.
In order to identify candidate membrane molecules of
the transport system,we employed two-phase partitioning
system which allows the isolation of cell membrane fraction
by a specific manner.Subsequently,we employed
high-resolution 2D electrophoresis and computer analysis
for detection of proteins with increased level in membrane
fraction of K562 cells under iron deprivation.The proteins
with increased level were identified employing liquid
chromatography combined with tandem mass spectrometry.
We have identified two such proteins,i.e.,aldolase A
(ALDA) and voltage-dependent anion channel 2 (VDAC2).
The increased expression of aldolase A and VDAC2 in
K562 cells under iron deprivation was confirmed by
western blot analysis.

英语翻译Ferric iron is reduced in a reaction with superoxide anion and subsequently ferrousiron is oxidized and thus iron can generate highly reactivehydroxyl radicals in Fenton reaction.The summary reaction,referred to as Haber-Weiss reaction,re
铁铁是减少的反应与超氧阴离子自由基,随后有色金属
铁氧化,从而铁可以产生高度反应
羟自由基在Fenton反应.总结反应,
被称为哈伯魏斯反应,结果在
羟自由基的生产和减少超
浓度[ 15 ] .低分子量铁配合
也能够产生活性氧[ 16 ,17 ] .
我们已经证明以前铁剥夺
刺激从铁摄取柠檬酸铁人红
K562细胞,这种刺激取决于
蛋白质的合成.然而,没有参与任何已知的
与蛋白质的作用,建议在非转铁运输
在质膜检测[ 18 ] .存在
交通运输系统的铁摄取低分子量
园区包括柠檬酸铁是有案可稽[ 19 -
23 ] .然而,具体的分子参与运输
系统仍然掩盖,尽管这一事实,即鉴定
这些分子是非常具有挑战性的主要原因
他们的潜力参与一些铁紊乱.
为了确定候选人分子膜
运输系统,我们采用两阶段划分
系统,使孤立细胞膜分数
一个特定的方式进行.随后,我们雇用
高分辨率二维电泳和计算机分析
检测的蛋白质水平增加膜
分数下K562细胞铁剥夺.这些蛋白质
提高水平,确定采用液体
色谱结合串联质谱.
我们已经确定了两个这种蛋白质,即醛甲
（阿尔达）和电压依赖阴离子通道2 （ VDAC2 ） .
增加醛缩酶的表达和在VDAC2
K562细胞铁剥夺下证实了
免疫印迹分析.

铁铁在与过氧化物阴离子和后来亚铁的反应减少iron被氧化和因而电烙可能引起高度易反应 在Fenton反应的hydroxyl基础。 概略反应， 指Haber-Weiss反应，发生 在过氧化物的hydroxyl根本生产和减退
concentration [15]。 低分子大量铁复合体 能也的are导致ROS [16， 17]。
We以前展示了那铁剥夺
stimulates...

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铁铁在与过氧化物阴离子和后来亚铁的反应减少iron被氧化和因而电烙可能引起高度易反应 在Fenton反应的hydroxyl基础。 概略反应， 指Haber-Weiss反应，发生 在过氧化物的hydroxyl根本生产和减退
concentration [15]。 低分子大量铁复合体 能也的are导致ROS [16， 17]。
We以前展示了那铁剥夺
stimulates从铁枸橼酸盐的铁举起由人的erythroleukemia
K562细胞和那这刺激依靠
protein综合。 然而，没有的介入任何知道 与一个建议的角色的proteins在非铁传递酶铁运输
across查出了质膜[18]。 存在 铁举起的transport系统从低分子大量
complexes包括铁枸橼酸盐是有大量文件证明的[19–
23]。 然而，在运输介入的具体分子
system保持阴暗，即使证明
of这些分子高度富挑战性主要归结于 在一些铁混乱的their潜在的介入。
为了辨认候选人膜分子
the运输系统，我们使用了两相分成 允许细胞膜分数的隔离的system
by具体方式。 随后，我们使用了
high-resolution第2电泳法和计算机分析 蛋白质的for侦查与增加的水平的在膜 K562在铁剥夺之下的细胞的fraction。 蛋白质 增加的with平实被辨认了使用液体
chromatography与纵排质谱分析结合了。
We辨认了二这样蛋白质，即，二磷酸果糖酶A
(ALDA)和电压依赖阴离子渠道2 (VDAC2)。
The二磷酸果糖酶A和VDAC2增加的表示
K562在铁剥夺之下的细胞被证实了
western污点分析。

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楼上两个都是翻译机翻的
楼主也真小气，才5分，50分我就帮你翻译了
我以前就是搞这个的